Preliminary studies reveal conolidine may well inhibit specific ion channels, minimizing neuronal excitability and restricting discomfort signals. This mechanism is particularly relevant in neuropathic ache, wherever abnormal signaling brings about persistent distress. In addition, conolidine appears to impact G protein-coupled receptor (GPCR) pathways integral to discomfort perceptio... https://napoleons656yiq8.wikigdia.com/user